HGH used with T4 instead of T3
Written by DavID on . Posted in
August 28, 2017 at 7:36 am #2413DavIDParticipant
Hello there everyone. I would like to get an answer to my question if there’s someone who can help. Well, there are quite a few postings on the internet that are saying ‘urging’ the user to use the T4 instead of T3 with the HGH. so… is that so far proven to be a better combination? Please explain as I can’t actually understand. Thanks in advance for everything!August 28, 2017 at 8:14 am #2414SuchaterParticipant
Umm… what’s the rationale for it??
In the time that the studies are suggesting that the GH might increase to T4 to T3 conversion… the studies I am talking about are:
1) on the ncbi pubmed search for *The influence of growth hormone and thyroxine on iodothyronine deiodinase activity in the liver, kidney and brown adipose tissue in hypophysectomized rats.*
2) on the healio com search for * GH therapy alters circulating thyroid hormones, deiodinase activity in men*
3) and on the sciencedirect com search for * Growth hormone acutely decreases type III iodothyronine deiodinase in chicken liver*
Start reading and you’re going to see… it is only seems to happen in lab animals and men with hypopituitarism, however not in normal men who do not need any GH… to be honest, I personally, so far, haven’t seen absolutely any proof of the GH users getting high blood T3 levels and full blown hyperthyroidism symptoms.
The OTOH androgens do increase the said T4 to T3 conversion a little bit. Hopefully this is going to give an answer to your question.August 28, 2017 at 8:36 am #2415DavIDParticipant
So well, this is not actually a study, however an article, an article on think steroids and it is named: ‘Thyroid Hormone + Growth Hormone – If You Aren’t Using T4 with Your GH, You’re Not Doing It Right’ and it is posted back in 2006.
I recommend you to read the entire article but the most important that I wanted to highline here is:
‘but we actually want the CONVERSION process of T4 into T3 to take place, because it’s the presence of those mediator enzymes that will allow the T3 to be synergistic with GH, instead of being inhibitory as is seen when T3 is simply added to a GH cycle. And remember, we don’t only want T3 levels high, but we want types 1 and 2 deiodinase to get us there- and when we take supplemental T3, that just doesn’t happen…’
This has got me thinking and that’s especially for that reason that there are absolutely no other sites or other specialists out there that I have found making this same claim I said…. However, this guy, still, in theory does sounds as if he knew what is he talking about… what do you think about this? does it sounds to be true? please read the entire article if you’re interested. If you can’t find it I am going to help. Write me privately. And yeah, by the way… I am not very sure if you are into HGH research or whatever… however it is quite common to use the thyroid hormone along with the HGH, however I still know that most people are taking the active form T3 over the T4 and that’s in the time that the rationale of Anthony it is that the conversion process, that is being accelerated by HGH, in itself has some benefits too.August 28, 2017 at 8:50 am #2416SCANASTParticipant
News flash this same opinion has been posted on the Meso quite a lot of times already. I’m talking about what op shared here.August 29, 2017 at 8:29 am #2417LockDownParticipant
To be honest, I would really like to find out what it is your opinion on this topic. The reason is that I’ve been reading a few of your last posts and you do seem to have some extensive knowledge so your opinion would mean a lot for me. thanks.August 29, 2017 at 8:54 am #2418SuchaterParticipant
Where are all of those LOTS of people out there who are having thyroid issues from the excess of the deiodinase levels, getting acromegaly symptoms like the clockwork?
Well, this is never happening.. and that’s why, the T4/ t3/ GH it is only a theory proven wrong.August 30, 2017 at 6:55 am #2419SCANASTParticipant
I’m not very sure if that has been addressed to me, but since I was the last poster I assumed that it is and that’s why I think that you’ve asked for my opinion. If that’s so… well.. in the time that the supplemental GH necessitates enhanced thyroxine secretion only those with preexisting thyroid dysfunction, or maybe also those using some extremely high GH dosages (like for example starting with 10 IU and even more of pharma every single day), are being unable to accelerate endogenous production in order to meet the demand! The baseline thyroid function testing does provide the best and most reliable answer with respect to who, may benefit from the supplemental thyroxine.
The latter it is very and very important due to the overwhelming majority of people out there the use of the thyroxine as a GH ‘ancillary’ it is not only unwarranted, but it is also significantly increasing the CV side effects too, and that’s especially when they are being used for a lot of months that are being typical of the GH ‘cycling’.
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